N-Acetyl Semax Amidate (30mg)

N-Acetyl Semax Amidate (30mg) is a dual-modified synthetic analog of Semax, itself derived from the ACTH(4-10) fragment. The added N-terminal acetylation and C-terminal amidation are protease-resistance modifications intended to extend the compound’s functional stability in research settings compared to unmodified Semax. It is most frequently studied for its proposed role in BDNF signaling and neuroplasticity research models.